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Journal
Presentation
    （Last updated : 2023-04-26 13:32:36）
  Yasuda Kobun
   Department   Specialty Division  Department of Immunology, Basic Medicine Seminars
   Position  
■ Journal
1. Original article　 IgG and IgE collaboratively accelerate expulsion of Strongyloides venezuelensis in a primary infection. 2013/07
2. Original article　 Skin-specific expression of IL-33 activates group 2 innate lymphoid cells and elicits atopic dermatitis-like inflammation in mice 2013/08 Link
3. Other　 Contribution of IL-33 to Pulmonary Eosinophilia in Strongyloides venezuelensis-infected Mice 2013/11
4. Introduction　 Importance of both innate immunity and acquired immunity for rapid expulsion of S.venezzuelensis 2014/03
5. Research report　 Immediate-type contact hypersensitivity is reduced in interleukin-33 knockout mice 2014/05 Link
6. Original article　 Immediate-type contact hypersensitivity is reduced in interleukin-33 knockout mice. 2014/05
7. Original article　 Epithelial-derived nuclear IL-33 aggravates inflammation in the pathogenesis of reflux esophagitis. 2014/08
8. Original article　 Interleukin-1/-33 Signaling Pathways as Therapeutic Targets for Endometriosis. 2019/08
9. Original article　 Anti-interleukin-6 receptor antibody treatment ameliorates postoperative adhesion formation 2019/11
10. Original article　 Lung fibroblasts produce interleukin-33 in
response to stimulation by retinoblastoma-binding protein 9 via production of
prostaglandin E2 2020/06
11. Original article　 Thymic stromal lymphopoietin contributes to protection of mice from Strongyloides venezuelensis infection by CD4 + T cell-dependent and -independent pathways 2021/04
12. Original article　 IL-33-Induced Keratoconjunctivitis Is Mediated by Group 2 Innate Lymphoid Cells in Mice 2023/04
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■ Presentation
1. Contribution of IL-33-activated type II innate lymphoid cells to pulmonary eosinophilia in Strongyloides venezuelensis-infected mice  2013/01/10
2. IL-33 KO mice exhibit the reduced immediate type contact hypersensitivity  2013/05/11
3. Contribution of IL-33-activted type II innate lymphoid cells to pulmonary eosinophilia in Strongyloides venezyelensis-infected mice  2013/08/26
4. Role of IL-33 in innate type eosinophilic inflammation  2013/12/12
5. Skin-specific expression of IL-33 causes atopic dermatitis-like inflammation with activation of group 2 innate lymphoid cells in mice  2014/02/08
6. Esophageal epithelial-derived IL33 acts as an exaggerator of inflammation in the pathogenesis of GERD  2014/05/06
7. Skin-specific expression of IL-33 causes atopic dermatitis-like inflammation with activation of group 2 innate lymphoid cells in mice  2014/05/10
8. Regnase-1 degradation is crucial for interleukin-33ーand interleukin-25ーmediated ILC2 activaton  2019/08/02
9. Caspase-1/11 regulates IL-33 secretion and partly mediates the devolpment of atopic dermatitis-like inflammation in mice  2019/11/10
10. IκB kinase complex-dependent Regnase-1 degradation is crucial for interleukin-33-and interleukin-25-mediated group-2 innate lymphoid cell activation  2019/12/13
11. Induction of antigen-specific immune response by IL-33 as a mucosal adjuvant  2021/12/09
12. Contribution of TSLP to the protection against Strongyloides venezuelensis infetion in a CD4+ T cell-dependent and independent manner  2022/05/28
13. Modo of action analysis of IL-33 as a mucosal adjuvant  2022/12/07
14. Role of thymic stromal lymphopoietin in the protection against Strongyloides venezuelensis infeciton  2022/12/07
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